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Karl Ludwig Kahlbaum (1828–1899), a German psychiatrist, is widely recognized for his seminal work Catatonia or Tension Insanity, published 1874, in which he first conceptualized catatonia as a distinct psychomotor disorder. However, the observation of catatonic signs in the context of mental illness predates Kahlbaum’s formal classification. Understanding the history and development of different clinical concepts of catatonia before and after Kahlbaum is paramount for grasping the intricacies of current classification systems such as ICD-11 and DSM-5. Historical perspectives provide valuable insights into the evolution of diagnostic criteria and conceptualizations of catatonia over time, highlighting the diverse array of clinical presentations and the challenges inherent in defining and categorizing this complex syndrome. Further, by studying historical perspectives, clinicians and researchers gain insight into the diverse ways in which catatonia has been perceived, defined, and categorized throughout history. This chapter includes previous work on the topic of the history of catatonia as well as more recent systematic research in electronic historical archives. It addresses the origins of catatonic signs during the eighteenth century and how the concept of catatonia has been embedded in historical psychiatric literature of the nineteenth century.
This chapter provides a sympathetic portrayal of Carl Jung, without glossing over his shortcomings and transgressions. It also explores the backgrounds, mindset, and aspirations of Jung and Freud that led to their attraction to each other, and ultimately their conflicts and the near-disastrous dissolution of their partnership. The chapter also covers the influences Otto Gross, Jung’s multi-year struggles with a near-psychotic state, his process of recovery, his exploration of various mystic traditions on the one hand, and the overlap between his thoughts and quantum physics on the other. It ends with a brief description of Jung’s influences on various new age and modern self-help movements. The chapter also discusses Jung’s complicated relationships with Sabina Spielrein, Toni Wolff, and his wife, Emma Jung, linking his attractions and attachments to major “anima” figures in his adult life with his lonely childhood, the deprivation of maternal attention, and his disappointments with a weak father.
The history of schizotypal personality disorder (SPD) begins with the history of schizophrenia. Eugen Bleuler, in his initial description of schizophrenic illness, broadened Kraepelin's construct of dementia praecox to include what Bleuler termed latent schizophrenia, a less severe, non-psychotic presentation of schizophrenia. Empirical evidence that latent schizophrenia might share a common etiology with more severe schizophrenia was Bleuler's observation of a familial link between latent and chronic schizophrenia. Kretschmer published illustrative pedigrees demonstrating the occurrence of schizophrenia-like symptoms among the family members of schizophrenic individuals, and described in some detail the characteristics of what he called a schizoid temperament observed among some of the relatives of these patients. The body of family-genetic research in schizophrenia provides empirical evidence for the presence of a non-psychotic syndrome characterized by milder forms of the symptoms of chronic schizophrenia in some of the biological relatives of schizophrenic individuals.
Descriptions of schizotypic psychopathology have a long history in the clinical psychopathology literature. Kraepelin and Eugen Bleuler made note of what they termed latent schizophrenia, a form of personality aberration thought to be, in essence, a quantitatively less severe expression of schizophrenia. P. E. Meehl described what he believed to be the four fundamental signs and symptoms of schizotypy: cognitive slippage, interpersonal aversiveness, anhedonia, and ambivalence. According to Meehl's model, schizotypy, as a personality organization reflective of a latent liability for schizophrenia, can manifest itself behaviorally and psychologically in various degrees of clinical compensation. Finally, Meehl conjectures that the base rate of schizotaxia in the general population is approximately 10%. Meehl's base rate estimate is found on a dominant-gene formulation, which posits that every schizophrenic person must have a parent of schizotype namely, a parent carrying the schizophrenia producing genotype.
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